Highlights:
1. Our lab has recently demonstrated that the snoRNA HBII-52 that is not expressed by people with Prader- Willi syndrome regulates alternative splicing of the serotonin 5 HT2C receptor.
1. Our lab has recently demonstrated that the snoRNA HBII-52 that is not expressed by people with Prader- Willi syndrome regulates alternative splicing of the serotonin 5 HT2C receptor.
2. We have now identified more than eight other pre-mRNAs that are regulated by the snoRNA HBII-52. The project will aim to a) understand the mechanism of snoRNA action, b) identify targets for other snoRNAs from the Prader-Willi critical region, c) develop mechanisms to substitute the loss of snoRNAs .
3. We are looking for a post doc with previous experience in RNA to join our team that studies pre-mRNA splicing. More information about the research is found at http://www.stamms-lab.net/
4. The position is funded by the NIH until 2013.
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